DoctorPSL
Resident Surgery Nerd
- Joined
- May 11, 2026
- Posts
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Too many of you are stalling on the big three out of fear, or worse, jumping straight to dutasteride without a baseline. If you’re a Norwood 2 or higher and you aren’t on at least 1mg/day of finasteride, you’re essentially LDARing while your follicles undergo miniaturization.
Finasteride inhibits roughly 70% of serum DHT. If your donor area is thinning or the recession continues, move to dutasteride—but be aware the half-life is significantly longer, meaning side effects can persist longer if you react poorly. Topical minoxidil is non-negotiable for vascularization, but it’s a growth stimulant, not a DHT blocker. If you don't address the androgenic component, the minoxidil gains will evaporate the moment you miss a few days.
Don't buy into the fear-mongering regarding post-finasteride syndrome unless you have clear physiological markers. Most of you are just experiencing nocebo effect. Get your bloods checked, titrate your dosage, and stop being a subhuman with a receding hairline. This isn't medical advice.
Finasteride inhibits roughly 70% of serum DHT. If your donor area is thinning or the recession continues, move to dutasteride—but be aware the half-life is significantly longer, meaning side effects can persist longer if you react poorly. Topical minoxidil is non-negotiable for vascularization, but it’s a growth stimulant, not a DHT blocker. If you don't address the androgenic component, the minoxidil gains will evaporate the moment you miss a few days.
Don't buy into the fear-mongering regarding post-finasteride syndrome unless you have clear physiological markers. Most of you are just experiencing nocebo effect. Get your bloods checked, titrate your dosage, and stop being a subhuman with a receding hairline. This isn't medical advice.